[1]黄露露,张孟贤,于世英.细胞间网络连接在高级别胶质瘤中的作用研究进展[J].中国临床神经外科杂志,2022,27(01):51-52.[doi:10.13798/j.issn.1009-153X.2022.01.019]
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细胞间网络连接在高级别胶质瘤中的作用研究进展()
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《中国临床神经外科杂志》[ISSN:1009-153X/CN:42-1603/TN]

卷:
27
期数:
2022年01期
页码:
51-52
栏目:
综述
出版日期:
2022-01-15

文章信息/Info

文章编号:
1009-153X(2022)01-0051-02
作者:
黄露露 张孟贤 于世英
430030 武汉,华中科技大学同济医学院附属同济医院肿瘤科(黄露露、张孟贤、于世英)
关键词:
高级别胶质瘤细胞间网络连接作用机制
分类号:
R 739.41
DOI:
10.13798/j.issn.1009-153X.2022.01.019
文献标志码:
A

参考文献/References:

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[2]马辉辉,张帆,杨林,等. 高级别胶质瘤术后放疗联合替莫唑胺治疗临床分析[J]. 中华肿瘤防治杂志,2017,24(18): 1295-1300.
[3]Hatoum A, Mohammed R, Zakieh O. The unique invasiveness of glioblastoma and possible drug targets on extracellular matrix[J]. Cancer Manag Res, 2019, 11 1843-1855.
[4]Phillips SL, Williams CB, Zambrano JN, et al. Connexin 43 in the development and progression of breast cancer: What's the connection (Review)[J]? Int J Oncol, 2017, 51(4): 1005-1013.
[5]Polusani SR, Huang YW, Huang G, et al. Adipokines deregulate cellular communication via epigenetic repression of gap junction loci in obese endometrial cancer[J]. Cancer Res, 2019, 79(1): 196-208.
[6]Su M, Zhang Q. Deficiency of gap junction composed of connexin43 contributes to oxaliplatin resistance in colon cancer cells[J]. Oncol Lett, 2017, 14(3): 3669-3674.
[7]Broekman ML, Maas SLN, Abels ER, et al. Multidimensional communication in the microenvirons of glioblastoma[J]. Nat Rev Neurol, 2018, 14(8): 482-495.
[8]Carone C, Genedani S, Leo G, et al. In vitro effects of cocaine on tunneling nanotube formation and extracellular vesicle release in glioblastoma cell cultures[J]. J Mol Neurosci,2015, 55(1): 42-50.
[9]Gurke S, Barroso JF, Gerdes HH. The art of cellular communication:tunneling nanotubes bridge the divide[J]. Histochem Cell Biol, 2008, 129(5): 539-550.
[10]Jenkins RB, Kimmel DW, Moertel CA, et al. A cytogenetic study of 53 human gliomas[J]. Cancer Genet Cytogenet,1989, 39(2): 253-279.
[11]Osswald M, Jung E, Sahm F, et al. Brain tumour cells interconnect to a functional and resistant network[J]. Nature,2015, 528(7580): 93-98.
[12]Klumpp L, Sezgin EC, Skardelly M, et al. KCa3.1 channels and glioblastoma: in vitro studies[J]. Curr Neuropharmacol,2018, 16(5): 627-635.
[13]杜小林,王襄阳,穆军博,等. 胶质瘤大电导钙激活钾通道的研究进展[J]. 中国临床神经外科杂志,2019,24(6):378-380.
[14]Okolie O, Bago JR, Schmid RS, et al. Reactive astrocytes potentiate tumor aggressiveness in a murine glioma resection and recurrence model[J]. Neuro Oncol, 2016, 18(12):1622-1633.
[15]Weil S, Osswald M, Solecki G, et al. Tumor microtubes convey resistance to surgical lesions and chemotherapy in gliomas[J]. Neuro Oncol, 2017, 19(10): 1316-1326.
[16]Venkatesh HS, Morishita W, Geraghty AC, et al. Electrical and synaptic integration of glioma into neural circuits[J].Nature, 2019, 573(7775): 539-545.
[17]Venkataramani V, Tanev DI, Strahle C, et al. Glutamatergic synaptic input to glioma cells drives brain tumour progression [J]. Nature, 2019, 573(7775): 532-538.
[18]Barria A. Dangerous liaisons as tumour cells form synapses with neurons[J]. Nature, 2019, 573(7775): 499-501.
[19]Venkatesh HS, Johung TB, Caretti V, et al. Neuronal activity promotes glioma growth through neuroligin-3 secretion[J].Cell, 2015, 161(4): 803-816.

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备注/Memo

备注/Memo:
基金项目:国家自然科学基金(81772680)
通讯作者:于世英,E-mail:dreamrain_hl@163.com
更新日期/Last Update: 2022-01-18