[1]许双祥,邹雅雯 综述,陈劲草 审校.烟雾病行血管重建术后脑高灌注综合征的研究进展[J].中国临床神经外科杂志,2019,(04):251-253.[doi:10.13798/j.issn.1009-153X.2019.04.022]
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烟雾病行血管重建术后脑高灌注综合征的研究进展()
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《中国临床神经外科杂志》[ISSN:1009-153X/CN:42-1603/TN]

卷:
期数:
2019年04期
页码:
251-253
栏目:
论著
出版日期:
2019-04-27

文章信息/Info

文章编号:
1009-153X(2019)04-0251-03
作者:
许双祥邹雅雯 综述陈劲草 审校
430071 武汉,武汉大学中南医院神经外科(许双祥、邹雅雯、陈劲草
关键词:
烟雾病血管重建术脑高灌注综合征脑血管反应力脑血容量
分类号:
R 743; R 651.1+2
DOI:
10.13798/j.issn.1009-153X.2019.04.022
文献标志码:
A

参考文献/References:

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[2] Suzuki J, Takaku A. Cerebrovascular "moyamoya" disease. Disease showing abnormal net-like vessels in base of brain [J]. Arch Neurol, 1969, 20(3): 288-299.
[3] Kuroda S, Houkin K. Moyamoya disease: current concepts and future perspectives [J]. Lancet Neurol, 2008, 7(11): 1056-1066.
[4] Pandey P, Steinberg GK. Neurosurgical advances in the treatment of moyamoya disease [J]. Stroke, 2011, 42(11): 3304-3310.
[5] Yu J, Shi L, Guo Y, et al. Progress on complications of direct bypass for Moyamoya Disease [J]. Int J Med Sci, 2016, 13 (8): 578-587.
[6] Hayashi K, Horie N, Suyama K, et al. Incidence and clinical features of symptomatic cerebral hyperperfusion syndrome after vascular reconstruction [J]. World Neurosurg, 2012, 78 (5): 447-454.
[7] Fujimura M, Mugikura S, Kaneta T, et al. Incidence and risk factors for symptomatic cerebral hyperperfusion after super- ficial temporal artery-middle cerebral artery anastomosis in patients with moyamoya disease [J]. Surg Neurol, 2009, 71 (4): 442-447.
[8] Uno M, Nakajima N, Nishi K, et al. Hyperperfusion syn- drome after extracranial-intracranial bypass in a patient with moyamoya disease--case report [J]. Neurol Med Chir (Tokyo), 1998, 38(7): 420-424.
[9] Kaku Y, Iihara K, Nakajima N, et al. Cerebral blood flow and metabolism of hyperperfusion after cerebral revascula- rization in patients with moyamoya disease [J]. J Cereb Blood Flow Metab, 2012, 32(11): 2066-2075.
[10] Han JS, Abou-Hamden A, Mandell DM, et al. Impact of extracranial-intracranial bypass on cerebrovascular reac- tivity and clinical outcome in patients with symptomatic moyamoya vasculopathy [J]. Stroke, 2011, 42(11): 3047- 3054.
[11] Uchino H, Kuroda S, Hirata K et al. Predictors and clinical features of postoperative hyperperfusion after surgical reva- scularization for moyamoya disease: a serial single photon emission CT/positron emission tomography study [J]. Stroke, 2012, 43: 2610-2616.
[12] Kang HS, Kim JH, Phi JH, et al. Plasma matrix metallopro teinases, cytokines and angiogenic factors in moyamoya disease [J]. J Neurol Neurosurg Psychiatry, 2010, 81(6): 673-678.
[13] Fujimura M, Kaneta T, Mugikura S, et al. Temporary neuro- logic deterioration due to cerebral hyperperfusion after superficial temporal artery-middle cerebral artery anasto- mosis in patients with adult-onset moyamoya disease [J]. Surg Neurol, 2007, 67(3): 273-282.
[14] Fujimura M, Shimizu H, Mugikura S, et al. Delayed intrace- rebral hemorrhage after superficial temporal artery-middle cerebral artery anastomosis in a patient with moyamoya disease: possible involvement of cerebral hyperperfusion and increased vascular permeability [J]. Surg Neurol, 2009, 71(2): 223-227.
[15] Gesang DZ, Zhang D, Zhao JZ, et al. Laser Doppler flowme- ter study on regional cerebral blood flow in early stage after standard superficial temporal artery-middle cerebral artery bypass surgery for moyamoya disease [J]. Chin Med J (Engl), 2009, 122(20): 2412-2418.
[16] Chen Y, Xu W, Guo X, et al. CT perfusion assessment of Moyamoya syndrome before and after direct revasculariza- tion (superficial temporal artery to middle cerebral artery bypass) [J]. Eur Radiol, 2016, 26(1): 254-261.
[17] Fujimura M, Inoue T, Shimizu H, et al. Efficacy of prophy- lactic blood pressure lowering according to a standardized postoperative management protocol to prevent symptomatic cerebral hyperperfusion after direct revascularization sur- gery for moyamoya disease [J]. Cerebrovasc Dis, 2012, 33 (5): 436-445.
[18] Uchino H, Nakayama N, Kazumata K, et al. Edaravone reduces hyperperfusion-related neurological deficits in adult moyamoya disease: historical control study [J]. Stroke, 2016, 47(7): 1930-1932.
[19] Fujimura M, Niizuma K, Inoue T, et al. Minocycline prevents focal neurological deterioration due to cerebral hyperper- fusion after extracranial-intracranial bypass for moyamoya disease [J]. Neurosurgery, 2014, 74(2): 163-170.

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备注/Memo

备注/Memo:
(2018-03-04收稿,2018-05-04修回) 通讯作者:陈劲草,E-mail:chenjincao@hotmail.com
更新日期/Last Update: 2019-04-27