[1]袁凡恩 综述 陈谦学 审校.胶质瘤干细胞的研究进展[J].中国临床神经外科杂志,2016,(08):508-511.[doi:10.13798/j.issn.1009-153X.2016.08.025]
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胶质瘤干细胞的研究进展()
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《中国临床神经外科杂志》[ISSN:1009-153X/CN:42-1603/TN]

卷:
期数:
2016年08期
页码:
508-511
栏目:
论著
出版日期:
2016-08-18

文章信息/Info

文章编号:
1009-153X(2016)08-0508-04
作者:
袁凡恩 综述 陈谦学 审校
430060 武汉,武汉大学人民医院神经外科
通讯作者:陈谦学,E-mail:chenqx666@sohu.com
关键词:
胶质瘤肿瘤干细胞肿瘤微环境免疫治疗放化疗抵抗
分类号:
R 739.41
DOI:
10.13798/j.issn.1009-153X.2016.08.025
文献标志码:
A

参考文献/References:

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[3] Liu G, Yuan X, Zeng Z, et al. Analysis of gene expression and chemoresistance of CD133+ cancer stem cells in glio- blastoma [J]. Mol Cancer, 2006, 5: 67.
[4] Ye J, Wu D, Wu P, et al. The cancer stem cell niche: cross talk between cancer stem cells and their microenvironment [J]. Tumour Biol, 2014, 35(5): 3945-3951.
[5] Heddleston JM, Li Z, Mclendon RE, et al. The hypoxic mi- croenvironment maintains glioblastoma stem cells and pro- motes reprogramming towards a cancer stem cell phenotype [J]. Cell Cycle, 2009, 8(20): 3274-3284.
[6] Fan X, Khaki L, Zhu TS, et al. NOTCH pathway blockade depletes CD133-positive glioblastoma cells and inhibits growth of tumor neurospheres and xenografts [J]. Stem Cells, 2010, 28(1): 5-16.
[7] Gustafsson MV, Zheng X, Pereira T, et al. Hypoxia requires notch signaling to maintain the undifferentiated cell state [J]. Dev Cell, 2005, 9(5): 617-628.
[8] Calabrese C, Poppleton H, Kocak M, et al. A perivascular niche for brain tumor stem cells [J]. Cancer Cell, 2007, 11(1): 69-82.
[9] Zhu TS, Costello MA, Talsma CE, et al. Endothelial cells create a stem cell niche in glioblastoma by providing NOTCH ligands that nurture self-renewal of cancer stem- like cells [J]. Cancer Res, 2011, 71(18): 6061-6072.
[10] Soda Y, Marumoto T, Friedmann-Morvinski D, et al. Trans- differentiation of glioblastoma cells into vascular endothe- lial cells [J]. Proc Natl Acad Sci USA, 2011, 108(11): 4274- 4280.
[11] Yi L, Xiao H, Xu M, et al. Glioma-initiating cells: a predo- minant role in microglia/macrophages tropism to glioma [J]. J Neuroimmunol, 2011, 232(1-2): 75-82.
[12] Wu A, Wei J, Kong L Y, et al. Glioma cancer stem cells induce immunosuppressive macrophages/microglia [J]. Neuro Oncol, 2010, 12(11): 1113-1125.
[13] Esparza R, Azad TD, Feroze AH, et al. Glioblastoma stem cells and stem cell-targeting immunotherapies [J]. J Neuro- oncol, 2015, 123(3): 449-457.
[14] Vik-Mo E O, Nyakas M, Mikkelsen B V, et al. Therapeutic vaccination against autologous cancer stem cells with mRNA-transfected dendritic cells in patients with glio- blastoma [J]. Cancer Immunol Immunother, 2013, 62(9): 1499-1509.
[15] Brown CE, Starr R, Aguilar B, et al. Stem-like tumor-initia- ting cells isolated from IL13Ralpha2 expressing gliomas are targeted and killed by IL13-zetakine-redirected T Cells [J]. Clin Cancer Res, 2012, 18(8): 2199-2209.
[16] Zhu X, Prasad S, Gaedicke S, et al. Patient-derived glio- blastoma stem cells are killed by CD133-specific CAR T cells but induce the T cell aging marker CD57 [J]. Oncotarget, 2015, 6(1): 171-184.
[17] Patel AP, Tirosh I, Trombetta JJ, et al. Single-cell RNA-seq highlights intratumoral heterogeneity in primary glioblast- oma [J]. Science, 2014, 344(6190): 1396-1401.
[18] Kim Y, Kim E, Wu Q, et al. Platelet-derived growth factor receptors differentially inform intertumoral and intratumoral heterogeneity [J]. Genes Dev, 2012, 26(11): 1247-1262.
[19] Lathia JD, Mack SC, Mulkearns-Hubert EE, et al. Cancer stem cells in glioblastoma [J]. Genes Dev, 2015, 29(12): 1203-1217.

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更新日期/Last Update: 2016-08-19