参考文献/References:
[1] Jiang B, Wang WZ, Bao QJ, et al. Incidence and trend of stoke and its subtypes in China results from three cities [J]. Stoke, 2006, 37: 63-68
[2] Kim E, Kim HC, Lee S, et al. Dexmedetomidine confer neu-roprotection against transient global cerebral ischemia/reperfusion injury in rats by inhibiting inflammation throughinactivation of the TLR-4/NF-kB pathwat [J]. Neurosci Lett, 2017, 649: 20-27.
[3] De Bock M, Wang N, Leybaert L, et al. Endothilia calcium dynamics, connexin channels and blood-brain barrier function [J]. Prog Neurobiol, 2013, 108: 1-20.
[4] De Bock M, Culot M, Simon AM, et al. Connexins channels provide a target to manipulate brain endothelial calcium dynamic and blood-brain barrier permeability [J]. J Cere Blood Flow Metab, 2011, 31(9): 1942-1957.
[5] Chen W, Feng JG, Tong WS. Phosphorylation of astrocytic Cx43 by ERK1/2 impairs BBB in acute cerebral ischemia [J]. Cell Biosci, 2017, 7: 43.
[6] Chen W, Zheng P, Feng JG, et al. Phosphorylation of Cx43 induced by TBI promotes exosome release [J]. J Neurophy-siol, 2018, 119(1): 305-311.
[7] Denis JF, Pfenniger A, Kwak BR, et al. Cx40 controls endo-thelial activation by dampening NF-κB activation [J]. Oncotarget, 2017, 8(31): 50972-50986.
[8] Elfgang C, Eckert R, Willecke K, et al. Specific permeabi-lity and selective formation of gap junction channels in connexin-tranfected Hela cells [J]. J Cell Biol, 129: 805-817.
[9] Azarashvili T, Baburina Y, Reiser G，et al. Calcium-inducedpermeability transition in rat brain mitochondria is promotedby carbenoxolone through targeting connexin [J]. Am J Physiol Cell Ph, 2011, 300(3): C707-720.
[10] Ohsumi A, Nawashiro H, Shima K, et al. Temporal and spa-tial profile of phosphorylated connexin43 after traumatic brain injury in rats [J]. J Neurotrauma, 2010, 27: 1255-1263.
[11] Chen B, Sun L, Ma J, et al. Correlation between connexin and traumatic brain injury in patients [J]. Brain and Behav, 2017, 7: e00770.
[12] Frantseva MV, Kokarovtseva L, Perez Velazquez JL, et al. Specific gap junctions enhance the neuronal vulnerability to brain traumatic injury [J]. J Neurosci, 2002, 22(3): 644-653.
[13] Chen W, Guo YJ, Tong WS, et al. Involvement of Cx40 in theprotective effects of ginsenoside Rb1 against TBI [J]. Cell Mol Neurobiol, 2016, 36(7): 1057-1065.
[14] 叶新运，洪 涛. Cx43/Cx45异型缝隙连接通道参与试验性蛛网膜下腔出血后脑血管痉挛的实验研究[J]. 中风与神经疾病杂志，2012，29（3）：196-199.
[15] Jayakumar AR, Tong XY, Norenberg MD, et al. Activation of NF-kB mediates astrocyte swelling and brain edema in TBI [J]. J Neurotrauma, 2014, 31(14): 1249-1257.
相似文献/References:
[1]黄 坦 黄书岚 陈谦学.NaHS抑制Nox4 和 p22p hox的表达保护小鼠脑缺血再灌注损伤[J].中国临床神经外科杂志,2016,(09):545.[doi:10.13798/j.issn.1009-153X.2016.09.011]
HUANG Tan,HUANG Shu-lan,CHEN Qian-xue..NaHS induces neuroprotection against cerebral ischemia reperfusion injury by inhibiting Nox4 and p22phox expressions in cerebral tissues in mice[J].,2016,(05):545.[doi:10.13798/j.issn.1009-153X.2016.09.011]
[2]陈 伟,郑 平,任大斌,等.cAMP对大鼠颅脑损伤后神经功能及脑水肿的影响[J].中国临床神经外科杂志,2019,(04):226.[doi:10.13798/j.issn.1009-153X.2019.04.012]
CHEN Wei,ZHENG Ping,REN Da-Bin,et al.Role of inhibition of phosphorylated Cx43 by PKA pathway in treatment for traumatic brain injury[J].,2019,(05):226.[doi:10.13798/j.issn.1009-153X.2019.04.012]
[3]汪 雷 胡火军 马金阳 黄 松 董元训 王旭光 周有东.七叶皂苷钠对大鼠脑缺血再灌注损伤的保护作用[J].中国临床神经外科杂志,2021,26(08):616.[doi:10.13798/j.issn.1009-153X.2021.08.013]
WANG Lei,HU Huo-jun,MA Jin-yang,et al.Protective effect of sodium aescinate on adult rats after cerebral ischemia-reperfusion injury[J].,2021,26(05):616.[doi:10.13798/j.issn.1009-153X.2021.08.013]