参考文献/References:
[1] Lozano D, Gonzales-Portillo GS, Acosta S, et al. Neuroin-flammatory responses to traumatic brain injury: etiology, clinical consequences, and therapeutic opportunities [J]. Neuropsychiatr Dis Treat, 2015, 11: 97-106.
[2] Denes A, Thornton P, Rothwell NJ, et al. Inflammation and brain injury: acute cerebral ischaemia, peripheral and central inflammation [J]. Brain Behav Immun, 2010, 24: 708-723.
[3] Rostène W, Kitabgi P, Parsadaniantz SM. Chemokines: a new class of neuromodulator [J]? Nat Rev Neurosci, 2007, 8: 895-903.
[4] Réaux-Le Goazigo A, Van Steenwinckel J, Rostène W, et al.Current status of chemokines in the adult CNS [J]. Prog Neurobiol, 2013, 104: 67-92.
[5] Cédile O, Wlodarczyk A , Owens T. CCL2 recruits T cells into the brain in a CCR2-independent manner [J]. APMIS, 2017, 125: 945-956.
[6] 刘 颖，张淑珍，朱晓红，等. 高压氧治疗对创伤性脑损伤大鼠认知功能的影响[J]. 中国康复医学杂志，2017，（32）：1214-1219.
[7] He M, Dong H, Huang Y, et al. Astrocyte -derived CCL2 is associated with M1 activation and recruitment of cultured microglial cells [J]. Cell Physiol Biochem, 2016, 38(3): 859-870.
[8] Stamatovic SM, Shakui P, Keep RF, et al. Monocyte chemo-attractant protein-1 regulation of blood-brain barrier per-meability [J]. J Cereb Blood Flow Metab, 2005, 25: 593-606.
[9] Wang Y, Wei Y, Oguntaya S, et al. Cerebrospinal fluid CCL2is an early-response biomarker for blast overpressure wave-induced neurotrauma in rats [J]. J Neurotrauma, 2017, 34: 952-962.
[10] 刘 苏，孙李颖，吴勤峰，等. 趋化因子CCL2在重度颅脑损伤病人中的表达[J]. 重庆医学，2016，45（24）：3406-3408.
[11] Graber DJ, Costine BA, Hickey WF. Early inflammatory mediator gene expression in two models of traumatic brain injury: ex vivo cortical slice in mice and in vivo cortical impact in piglets [J]. J Neuroinflammation, 2015, 12: 76.
[12] Dzenko KA, Song L, Ge S, et al. CCR2 expression by brain microvascular endothelial cells is critical for macrophage transendothelial migration in response to CCL2[J]. Micro-vasc Res, 2005, 70: 53-64.
[13] Semple BD, Bye N, Rancan M, et al. Role of CCL2 (MCP-1)in traumatic brain injury (TBI): evidence from severe TBI patients and CCL2-/- mice [J]. J Cereb Blood Flow Metab, 2010, 30(4): 769-782.
[14] Liu S, Zhang L, Wu Q, et al. Chemokine CCL2 induces apoptosis in cortex following traumatic brain injury [J]. J Mol Neurosci, 2013, 51(3): 1021-1029.
[15] Semple BD, Frugier T, Morganti-Kossmann MC. CCL2 modulates cytokine production in cultured mouse astrocytes [J]. J Neuroinflammation, 2010, 7(1): 67.
[16] Sharif Y, Jumah F, Coplan L, et al. Blood brain barrier: a review of its anatomy and physiology in health and disease [J]. Clin Anat, 2018, 31(6): 812-823.
[17] 郭秀丽，王传宝，郭 蕾. 实验性自身免疫性脑脊髓炎大鼠紧密连接蛋白表达的变化研究[J]. 中华神经医学杂志，2018，17（8）：772-777.
[18] Szmydynger-Chodobska J, Strazielle N, Gandy JR, et al. Posttraumatic invasion of monocytes across the blood-cerebrospinal fluid barrier [J]. J Cereb Blood Flow Metab, 2012, 32(1): 93-104.
[19] Teixeira MM, Vilela MC, Soriani FM, et al. Using intravital microscopy to study the role of chemokines during infection and inflammation in the central nervous system [J]. J Neuroimmunol, 2010, 224(1-2): 62-65.
[20] Ge S, Song L, Serwanski DR, et al. Transcellular transport ofCCL2 across brain microvascular endothelial cells [J]. J Neurochem, 2008, 104(5): 1219-1232.
[21] Sagar D, Lamontagne A, Foss CA, et al. Dendritic cell CNS recruitment correlates with disease severity in EAE via CCL2 chemotaxis at the blood-brain barrier through paracellular transmigration and ERK activation[J]. J Neuro-inflammation, 2012, 9: 245.
[22] Bartholom?us I, Kawakami N, Odoardi F, et al. Effector T cell interactions with meningeal vascular structures in nascent autoimmune CNS lesions [J]. Nature, 2009, 462(7269): 94-98.
[23] Ruan L, Kang Z, Pei G, et al. Amyloid deposition and infla-mmation in APPswe/PS1dE9 mouse model of alzheimers disease [J]. Curr Alzheimer Res, 2009, 6(6): 531-540.
[24] Westin K, Buchhave P, Nielsen H, et al. CCL2 is associatedwith a faster rate of cognitive decline during early stages of alzheimer’s disease [J]. PloS One, 2012, 7: e30525.
[25] 贾雪琳，卢 红. MBP H-FABP MCP-1与脑梗死病情分期及预后的相关性分析[J]. 中国实用神经疾病杂志，2019，15：1662-1668.
[26] Fabene PF, Bramanti P, Constantin G. The emerging role for chemokines in epilepsy [J]. J Neuroimmunol, 2010, 224(1-2): 22-27.
[27] Madrigal JLM, Caso JR. The chemokine (C-C motif) ligand 2 in neuroinflammation and neurodegeneration [J]. Adv Exp Med Biol, 2014, 824: 209-219.
[28] Ho L, Zhao W, Dams-O’Connor K, et al. Elevated plasma MCP-1 concentration following traumatic brain injury as a potential "predisposition"factor associated with an increased risk for subsequent development of Alzheimer’s disease [J]. J Alzheimers Dis, 2012, 31(2): 301-313.
[29] Huang XJ, Glushakova O, Mondello S, et al. Acute temporalprofiles of serum levels of UCH-L1 and GFAP and relation-ships to neuronal and astroglial pathology following trauma-tic brain injury in rats [J]. J Neurotrauma, 2015, 32(16): 1179-1189.
[30] Di Pietro V, Amorini AM, Lazzarino G, et al. S100B and glial fibrillary acidic protein as indexes to monitor damage severity in an in vitro model of traumatic brain injury [J]. Neurochem Res, 2015, 40(5): 991-999.
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