[1]李庆勇 综述 钱志远 审校.基质金属蛋白酶与颅脑损伤研究进展[J].中国临床神经外科杂志,2015,(05):312-314.[doi:10.13798/j.issn.1009-153X.2015.05.021]
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基质金属蛋白酶与颅脑损伤研究进展()
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《中国临床神经外科杂志》[ISSN:1009-153X/CN:42-1603/TN]

卷:
期数:
2015年05期
页码:
312-314
栏目:
论著
出版日期:
2015-05-30

文章信息/Info

文章编号:
1009-153X(2015)05-0312-03
作者:
李庆勇 综述 钱志远 审校
215004 江苏苏州,苏州大学附属第二医院神经外科
关键词:
颅脑损伤基质金属蛋白酶神经血管单元血管再生轴突再生
分类号:
R 651.1+5
DOI:
10.13798/j.issn.1009-153X.2015.05.021
文献标志码:
B

参考文献/References:

[1] 沈合春,钱志远. 神经血管单元与颅脑损伤[J]. 中华创伤 杂志,2010,26(10):957-960.
[2] Milward EA, Fitzsimmons C, Szklarczyk A, et al. The matrix metalloproteinases and CNS plasticity: an overview [J]. J Neuroimmunol, 2007, 187: 9-19.
[3] Candelario-Jalil E, Yang Y, Rosenberg GA. Diverse roles of matrix metalloproteinases and tissue inhibitors of metallo- proteinases in neuroinflammation and cerebral ischemia [J]. Neuroscience, 2009, 158: 983-994.
[4] Heo JH, Lucero J, Abumiya T, et al. Matrix metalloprotein- ases increase very early during experimental focal cerebral ischemia [J]. J Cereb Blood Flow Metab, 1999, 19(6): 624- 633.
[5] Von Gertten C, Holmin S, Mathiesen T, et al. Increases in matrix metalloproteinase-9 and tissue inhibitor of matrix metalloproteinase-1 mRNA after cerebral contusion and depolarisation [J]. J Neurosci Res, 2006, 73(6): 803-810.
[6] Chen ZL, Indyk JA, Stricldand S. The hippocampal lanmi- nin matrix is dynamical critical for neuronal survival [J]. Mol Biol Cell, 2003, 14(7): 2665-2676.
[7] Gessler M, Gottschling C, Wetzel CH, et al. Primary hippo- campal neurons, which lack four crucial extracellular ma- trix molecules, display abnormalities of synaptic structure and function and severe deficits in perineuronal net forma- tion [J]. J Neurosci, 2013, 33(18): 7742-7755.
[8] An P, Xue YX. Effects of preconditioning on tight junction and cell adhesion of cerebral endothelial cells[J]. Brain Res, 2009, 1272: 81-88.
[9] Chodobski A, Zink BJ, Szmydynger-Chodobska J. Blood- brain barrier pathophysiology in traumatic brain injury [J]. Transl Stroke Res, 2011, 2(4): 492-516.
[10] Walmsley AR, Mir AK. Targeting the Nogo-A signalling pathway to promote recovery following acute CNS injury [J]. Curr Pharm Res, 2007, 13(24): 2470-2484.
[11] Polikov VS, Block ML, Fellous JM, et al. In vitro model of glial scarring around neuroelectrodes chronically [J]. Bio- materials, 2006, 27: 5368-5376.
[12] Larsen PH, Wells JE, Stallcup WB, et al. Matrix metallopro- teinase-9 facilitates remyelination in part by processing the inhibitory NG2 proteoglycan [J]. J Neurosci, 2003, 23: 11127-11135.
[13] Ayoub AE, Cai TQ, Kaplan RA, et al. Developmental ex- pression of matrix metalloproteinases 2 and 9 and their po- tential role in the histogenesis of the cerebellar cortex [J]. J Comp Neurol, 2005, 481: 403-415.
[14] Vaillant C, Meissirel C, Mutin M, et al. MMP-9 deficiency affects axonal outgrowth, migration, and apoptosis in the developing cerebellum [J]. Mol Cell Neurosci, 2003, 24: 395-408.
[15] Quaegebeur A, Lange C, Carmeliet P. The neurovascular link in health and disease: molecular mechanisms and therapeutic implications [J]. Neuron, 2011, 71: 406-424.
[16] Matter K, Balda MS. Functional analysis of tight junctions [J]. Methods, 2003, 30(3): 228-234.
[17] Pepper MS. Role of the matrix metalloproteinase and plas- minogen activator-plasmin systems in angiogenesis [J]. Arterioscler Thromb Vasc Biol, 2001, 21(7): 1104-1117.
[18] van Hinsbergh VW, Engelse MA, Quax PH. Pericellular proteases in angiogenesis and vasculogenesis [J]. Arterio- scler Thromb Vasc Biol, 2006, 26(4): 716-728.

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更新日期/Last Update: 2015-05-30