参考文献/References:
[1] Sangiorgi S, De Benedictis A, Protasoni M, et al. Early-
stage microvascular alterations of a new model of controlled
cortical traumatic brain injury: 3D morphological analysis
using scanning electron microscopy and corrosion casting
[J]. J Neurosurg, 2013, 118(4): 763-774.
[2] Schwarzmaier SM, Gallozzi M, Plesnila N. Identification of
the vascular source of vasogenic brain edema following
traumatic brain injury using in vivo 2-photon microscopy in
mice [J]. J Neurotrauma, 2015, 32(13): 990-1000.
[3] Lou N, Takano T, Pei Y, et al. Purinergic receptor P2RY12-
dependent microglial closure of the injured blood-brain
barrier [J]. Proc Natl Acad Sci USA, 2016, 113: 1074-1079.
[4] Giza CC, Hovda DA. The new neurometabolic cascade of
concussion [J]. Neurosurgery, 2014, 75: S24-S33.
[5] Hinson HE, Rowell S, Schreiber M. Clinical evidence of
inflammation driving secondary brain injury: a systematic
review [J]. J Trauma Acute Care Surg, 2015, 78: 184-191.
[6] Chen JQ, Zhang CC, Jiang SN, et al. Effects of aquaporin 4
knockdown on brain edema of the uninjured side after
traumatic brain injury in rats[J]. Med Sci Monitor, 2016, 22:
4809-4819.
[7] 易 勇,周章明,向 前,等. 创伤型颅脑损伤患者血清及
脑组织中水通道蛋白1的表达水平及意义[J]. 标记免疫
分析与临床,2018,25(4):537-541.
[8] Huria T, Beeraka NM, Al-Ghamdi B, et al. Premyelinated
central axons express neurotoxic NMDA receptors: rele-
vance to early developing white-matter injury [J]. J Cereb
Blood Flow Metab, 2015, 35(4): 543-553.
[9] Chung C, Marson JD, Zhang QG, et al. Neuroprotection
mediated through GluN2C-Containing N-methyl-D-
aspartate (NMDA) receptors following ischemia [J]. Sci
Rep, 2016, 6: 37033:1-10.
[10] Cattani D, de Liz Oliveira Cavalli VL, Heinz Rieg CE, et al.
Mechanisms underlying the neurotoxicity induced by gly-
phosate-based herbicide in immature rat hippocampus:
involvement of glutamate excitotoxicity [J]. Toxicology,
2014, 320: 34-45.
[11] Sachser RM, Santana F, Crestani AP, et al. Forgetting of
long-term memory requires activation of NMDA receptors,
L-type voltage-dependent Ca2+ channels, and calcineurin
[J]. Sci Rep, 2016, 6: 22771.
[12] Lai TW, Zhang S, Wang YT. Excitotoxicity and stroke:
identifying novel targets for neuroprotection [J]. Prog
Neurobiol, 2014, 115: 157-188.
[13] Molinaro P, Cataldi M, Cuomo O, et al. Genetically modified
mice as a strategy to unravel the role played by the Na(+)/
Ca(2+) exchanger in brain ischemia and in spatial learning
and memory deficits [J]. Adv Exp Med Biol, 2013, 961:
213-222.
[14] Uchino H, Ogihara Y, Fukui H, et al. Brain injury following
cardiac arrest: pathophysiology for neurocritical care [J]. J
Intensive Care, 2016, 4: 31.
[15] Cornelius C, Crupi R, Calabrese V, et al. Traumatic brain
injury: oxidative stress and neuroprotection [J]. Antioxid
Redox Signal, 2013, 19(8): 836-853.
[16] Hill RL, Singh IN, Wang JA, et al. Time courses of post-
injury mitochondrial oxidative damage and respiratory
dysfunction and neuronal cytoskeletal degradation in a rat
model of focal traumatic brain injury [J]. Neurochem Int,
2017, 111: 45-56.
[17] Wong VS, Langley B. Epigenetic changes following trauma-
tic brain injury and their implications for outcome, recovery
and therapy [J].Neurosci Lett, 2016, 625: 26-33.
[18] Mendes Arent A, Souza LFd, Walz R, et al. Perspectives on
molecular biomarkers of oxidative stress and antioxidant
strategies in traumatic brain injury [J]. Biomed Res Int,
2014, 2014: 1-18.
[19] Bains M, Hall ED. Antioxidant therapies in traumatic brain
and spinal cord injury [J]. Biochim Biophys Acta, 2012,
1822(5): 675-684.
[20] Ahn SM, Kim HN, Kim YR, et al. Neuroprotective effect of
1-methoxyoctadecan-1-ol from Uncaria sinensis on
glutamate-induced hippocampal neuronal cell death [J]. J
Ethnopharmacol, 2014, 155(1): 293-299.
[21] Kumar A, Loane DJ. Neuroinflammation after traumatic
brain injury: opportunities for therapeutic intervention [J].
Brain Behav Immun, 2012, 26(8): 1191-1201.
[22] 李 峰,田冰锋,魏小兵,等. 重型颅脑创伤急性期和慢性
期外周血炎症反应标志物和氧化应激临床研究[J]. 中国
现代神经疾病杂志,2018,18(2):128-133.
[23] Hickman SE, Kingery ND, Ohsumi TK, et al. The microglial
sensome revealed by direct RNA sequencing [J]. Nat
Neurosci, 2013, 16(12): 1896-1905.
[24] Corps KN, Roth TL, McGavern DB. Inflammation and
neuroprotection in traumatic brain injury [J]. JAMA Neurol,
2015, 72(3): 355-362.
[25] Kelso ML, Gendelman HE. Bridge between neuroimmunity
and traumatic brain injury [J]. Curr Pharm Des, 2014, 20
(26): 4284-4298.
[26] Palta S, Saroa R, Palta A. Overview of the coagulation
system [J]. Indian J Anaesth, 2014, 58(5): 515-523.
[27] 李志伟,王著军,徐 旭,等. TNF-α、LPS、IL-6、PAT与颅
脑损伤后急性凝血功能障碍的相关性[J]. 中国临床神经
外科杂志,2015,20(1):25-27.
相似文献/References:
[1]张善纲 综述.现代战争中颅脑损伤的特点及功能康复[J].中国临床神经外科杂志,2015,(11):701.[doi:10.13798/j.issn.1009-153X.2015.11.021]
[2]钟秀均 黄书岚.复方醒脑液治疗颅脑损伤后综合征的临床观察[J].中国临床神经外科杂志,2016,(05):278.[doi:10.13798/j.issn.1009-153X.2016.05.007]
ZHONG Xiu-jun,HUANG Shu-lan..Clinical observation on treatment of compound decoction for cerebral prorection for post-traumatic brain syndrome[J].,2016,(12):278.[doi:10.13798/j.issn.1009-153X.2016.05.007]
[3]李国亮 邸 方 杨亚东.香芹酚通过抑制脑水肿与氧化应激反应保护大鼠
颅脑损伤[J].中国临床神经外科杂志,2016,(05):283.[doi:10.13798/j.issn.1009-153X.2016.05.009]
LI Guo-liang,DI Fang,YANG Ya-dong..Carvacrol protects against traumatic brain injury in rats through suppressing the brain edma and oxidative stress[J].,2016,(12):283.[doi:10.13798/j.issn.1009-153X.2016.05.009]
[4]罗心凯 陈治标 陈谦学.免缝胶原海绵人工硬脑膜在颅脑损伤大骨瓣减压术中的应用[J].中国临床神经外科杂志,2016,(06):357.[doi:10.13798/j.issn.1009-153X.2016.06.012]
LUO Xin-kai,CHEN Zhi-biao,CHEN Qian-xue..Application of collagen sponge artificial dura without suturing to big bone flap decompression after traumatic brain injury[J].,2016,(12):357.[doi:10.13798/j.issn.1009-153X.2016.06.012]
[5]贺瑛福 肖宗宇 马进海 许常林 裴 杰 袁 岗 张广华 巨 虎.高海拔地区重型颅脑损伤血清FSH、LH、PRL的变化[J].中国临床神经外科杂志,2016,(06):359.[doi:10.13798/j.issn.1009-153X.2016.06.013]
HE Ying-fu,XIAO Zong-yu,MA Jin-hai,et al.Changes in serum FSH, LH and PRL levels in the patients with severe traumatic brain injury in plateau[J].,2016,(12):359.[doi:10.13798/j.issn.1009-153X.2016.06.013]
[6]满明昊 李立宏 杨彦龙 李 敏 郭少春.颅脑损伤术后细胞免疫指标变化及脾多肽对其影响[J].中国临床神经外科杂志,2016,(06):362.[doi:10.13798/j.issn.1009-153X.2016.06.014]
MAN Ming-hao,LI Li-hong,YANG Yan-yong,et al.Changes in peripheral cells immunity after operation and effect of spleen polypeptide on them in patients with traumatic brain injury[J].,2016,(12):362.[doi:10.13798/j.issn.1009-153X.2016.06.014]
[7]程 勇 秦加新 戢翰升 李 刚 聂劲林 李 强.颅脑损伤术中急性脑膨出的相关因素分析[J].中国临床神经外科杂志,2016,(06):374.[doi:10.13798/j.issn.1009-153X.2016.06.019]
[8]张一帆 综述 张国来 审校.弥漫性轴索损伤影像学诊断的研究进展[J].中国临床神经外科杂志,2016,(07):442.[doi:10.13798/j.issn.1009-153X.2016.07.020]
[9]陈吉钢 张丹枫 魏嘉良 邹 伟 侯立军.创伤性眶上裂综合征的治疗陈吉钢[J].中国临床神经外科杂志,2016,(04):200.[doi:10.13798/j.issn.1009-153X.2016.04.003]
CHEN Ji-gang,ZHANG Dan-feng,WEI Jiang-liang,et al.Management of traumatic superior orbital fissure syndrome (report of 22 cases)[J].,2016,(12):200.[doi:10.13798/j.issn.1009-153X.2016.04.003]
[10]肖连福 黄瑞宏 陈昌勇.外伤性颅内静脉窦血栓形成1例[J].中国临床神经外科杂志,2016,(04):256.[doi:10.13798/j.issn.1009-153X.2016.04.024]
[11]王雅微综述,冯杰审校.PAI-1与颅脑损伤相关性的研究进展[J].中国临床神经外科杂志,2022,27(12):1032.[doi:10.13798/j.issn.1009-153X.2022.12.026]