[1]孙连杰,麦麦提依明·托合提,杨小朋.与胶质瘤诊断及预后相关的分子标记物的研究进展[J].中国临床神经外科杂志,2018,(09):633-635.[doi:10.13798/j.issn.1009-153X.2018.09.022]
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与胶质瘤诊断及预后相关的分子标记物的研究进展()
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《中国临床神经外科杂志》[ISSN:1009-153X/CN:42-1603/TN]

卷:
期数:
2018年09期
页码:
633-635
栏目:
论著
出版日期:
2018-09-25

文章信息/Info

文章编号:
1009-153X(2018)09-0633-03
作者:
孙连杰麦麦提依明·托合提杨小朋
830001 乌鲁木齐,石河子大学医学院研究生院(孙连杰);830000 乌鲁木齐,新疆维吾尔自治区人民医院神经外科(麦麦提依明﹒托合提、杨小朋)
关键词:
胶质瘤诊断预后分子标记物
分类号:
R 739.41
DOI:
10.13798/j.issn.1009-153X.2018.09.022
文献标志码:
A

参考文献/References:

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[2] 《中国中枢神经系统胶质瘤诊断和治疗指南》编写组. 中 国中枢神经系统胶质瘤诊断与治疗指南(2015)[J]. 中华 医学杂志,2016,96(7):485-509.
[3] Parsons DW, Jones S, Zhang X, et al. An integrated genomic analysis of human glioblastoma multiforme [J]. Science, 2008, 321: 1807-1812.
[4] Visani M, Acquaviva G, Marucci G, et al. Non-canonical IDH1 and IDH2 mutations: a clonal and relevant event in an Italian cohort of gliomas classified according to the 2016 World Health Organization (WHO) criteria[J]. J Neurooncol, 2017, 135(2): 245-254.
[5] Hartmann C, Hentschel B, Wick W, et al. Patients with IDH1 wild type anaplastic astrocytomas exhibit worse prognosis than IDH1-mutated glioblastomas, and IDH1 mutation status accounts for the unfavorable prognostic effect of higher age: implications for classification of gliomas [J]. Acta neuropathol, 2010, 120(6): 707-718.
[6] Noushmehr H, Weisenberger DJ, Diefes K, et al. Identifica- tion of a CpG island methylator phenotype that defines a distinct subgroup of glioma [J]. Cancer Cell, 2010, 17(5): 510-522.
[7] Cen L, Carlson BL, Schroeder MA, et al. p16-Cdk4-Rb axis controls sensitivity to a cyclin-dependent kinase inhibitor PD0332991 in glioblastoma xenograft cells [J]. Neuro Oncol, 2012, 14(7): 870-881.
[8] Juratli TA, Lautenschl?ger T, Geiger KD, et al. Radio- chemotherapy improves survial in IDH-mutant, 1p/19q non-codeleted secondary high-grade astrocytoma patients [J]. J Neurooncol, 2015, 124(2): 197-205.
[9] Wang L, Li Z, Liu C, et al. Comparative assessment of three methods to analyze MGMT methylation status in a series of 350 gliomas and gangliogliomas [J]. Pathol Res Pract, 2017, 213(12): 1489-1493.
[10] Lee A, Youssef I, Osborn VW, et al. The utilization of MGMT promoter methylation testing in United States hospitals for glioblastoma and its impact on prognosis [J]. J Clin Neurosci, 2018, 51(5): 85-90.
[11] Reifenberger J, Reifenberger G, Liu L, et al. Molecular genetic analysis of oligodendroglial tumors shows preferen- tial allelic deletions on 19q and 1p [J]. Am J Pathol, 1994, 145(5): 1175-1190.
[12] 崔向丽,赵志刚,任晓辉,等. 胶质瘤染色体1p/19q联合 缺失特点分析[J]. 中华外科杂志,2010,48(11):852-855.
[13] van den Bent MJ, Weller M, Wen PY, et al. A clinical pers- pective on the 2016 WHO brain tumor classification and routine molecular diagnostics [J]. Neuro Oncol, 2017, 19(5): 614-624.
[14] Killela PJ, Reitman ZJ, Jiao Y, et al. TERT promoter muta- tions occur frequently in gliomas and a subset of tumors derived from cells with low rates of self renewal [J]. Proc Natl Acad Sci USA, 2013, 10(15): 6021-6026.
[15] Eckel-Passow JE, Lachance DH, Molinaro AM, et al. Glioma groups based on 1p/19q, IDH, and TERT promoter mutations in tumors [J]. N Engl J Med, 2015, 372(26): 2499-2508.
[16] Labussiere M, Di Stefano AL, Gleize V, et al. TERT promo- ter mutations in gliomas, genetic associations and clinico- pathological correlations [J]. Br J Cancer, 2014, 111(10): 2024-2032.
[17] Kosty J, Lu F, Kupp R, et al. Harnessing OLIG2 function in tumorigenicity and plasticity to target malignant gliomas [J]. Cell Cycle, 2017, 16(8): 1654-1660.
[18] Durand K S, Guillaudeau A, Weinbreck N, et al. 1p19q LOH patterns and expression of p53 and Olig2 in gliomas, relation with histological types and prognosis [J]. Modern Pathol, 2010, 23(4): 619-628.
[19] Takami H, Yoshida A, Fukushima S, et al. Revisiting TP53 mutations and immunohistochemistry-a comparative study in 157 diffuse gliomas [J]. Brain Pathol, 2015, 25: 256-265.
[20] Yao TW, Zhang J, Prados M, et al. EGFR blockade prevents glioma escape from BRAFV600E, targeted therapy [J]. Oncotarget, 2015, 6(26): 21993-22005.
[21] Stec W, Rosiak K, Treda C, et al. Cyclictrans- phosphory- lation in a homodimer as the predominant mechanism of EGFRvⅢ action and regulation [J]. Oncotarget, 2018, 9(9): 8560-8572.
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备注/Memo

备注/Memo:
基金项目:新疆维吾尔自治区自然科学基金(2017D01C146) 通讯作者:杨小朋,E-mail:yxp9972 @sina.com
更新日期/Last Update: 2018-09-30