[1]陆丽娟,李玉凤,赵喜庆 综述,等.氯离子通道与胶质瘤的关系研究进展[J].中国临床神经外科杂志,2016,(12):803-805.[doi:10.13798/j.issn.1009-153X.2016.12.029]
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氯离子通道与胶质瘤的关系研究进展()
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《中国临床神经外科杂志》[ISSN:1009-153X/CN:42-1603/TN]

卷:
期数:
2016年12期
页码:
803-805
栏目:
论著
出版日期:
2016-12-25

文章信息/Info

文章编号:
1009-153X(2016)12-0803-03
作者:
陆丽娟李玉凤赵喜庆 综述胡万宁 审校
063000 河北唐山,华北理工大学附属唐山市人民医院(陆丽娟,李玉凤,赵喜庆,胡万宁)
关键词:
胶质瘤氯离子通道发病机制
分类号:
R 739.41
DOI:
10.13798/j.issn.1009-153X.2016.12.029
文献标志码:
A

参考文献/References:

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[2] Stobrawa SM, Breiderhoff T, Takamori S, et al. Disruption ofClC-3, a chloride channel expressed on synaptic vesicles, leads to a loss of the hippocampus [J]. Neuron, 2001, 29(1): 185-196.
[3] McFerrin MB, Sontheimer H. A role for ion channels in gli-oma cell invasion [J]. Neuron Glia Biol, 2006, 2(1): 39-49.
[4] Ransom CB, O’Neal JT, Sontheimer H. Volume-activated chloride currents contribute to the resting conductance and invasive migration of human glioma cells [J]. J Neurosci,2001, 21(19): 7674-7683.
[5] Habela CW, Ernest NJ, Swindall AF, et al. Chloride accu-mulation drives volume dynamics underlying cell prolifera-tion and migration [J]. J Neurophysiol, 2009, 101(2): 750-757.
[6] Habela CW, Sontheimer H. Cytoplasmic volume condensa-tion is an integral part of mitosis [J]. Cell Cycle, 2007, 6(13):1613-1620.
[7] Habela CW, Olsen ML, Sontheimer H. ClC3 is a critical regulator of the cell cycle in normal and malignant glial cells [J]. J Neurosci, 2011, 28(37): 9205-9217.
[8] Schwab A, Fabian A, Hanley PJ, et al. Role of ion channelsand transporters in cell migration [J]. Physiol Rev, 2012, 92(4): 1865-1913.
[9] Watkins S, Sontheimer H. Hydrodynamic cellular volume changes enable glioma cell invasion [J]. J Neurosci, 2011, 31(47): 17250-17259.
[10] Luigi C, Francesco A, Bernard F, et al. Serum-activated K and Cl currents underlay U87-MG glioblastoma cell migra-tion [J]. J Cell Physiol, 2011, 226(7): 1926-1933.
[11] Cuddapah VA, Sontheimer H. Molecular interaction and functional regulation of ClC-3 by Ca2+/calmodulin-depen-dent protein kinaseⅡ (CaMKⅡ) in human malignant glioma[J]. J Biol Chem, 2010, 285(15): 11188-11196.
[12] Sontheimer H. Ion channels and amino acid transporters support the growth and invasion of primary brain tumors [J]. Mol Neurobiol, 2004, 29(1): 61-71.
[13] Cuddapah VA, Turner KL, Seifert S, et al. Bradykinin-induced chemotaxis of human gliomas requires the activa-tion of K(Ca)3.1 and ClC-3 [J]. J Neurosci, 2013, 33(4): 1427-1440.
[14] Mcferrin MB, Turner KL, Cuddapah VA, et al. Differential role of IK and BK potassium channels as mediators of in-trinsic and extrinsic apoptotic cell death [J]. Ajp Cell Phy-siology, 2012, 303(10): 1070-1078.
[15] Ernest NJ, Habela CW, Sontheimer H. Cytoplasmic conden-sation is both necessary and sufficient to induce apoptotic cell death [J]. J Cell Sci, 2008, 121(3): 290-297.
[16] Weaver AK, Liu X, Sontheimer H. Role for calcium-activa-ted potassium channels (BK) in growth control of human malignant gliomacells [J]. J Neurosci Res, 2004, 78(2): 224-234.
[17] Deshane J, Garner CC, Sontheimer H. Chlorotoxin inhibits glioma cell invasion via matrix metalloproteinase-2 [J]. J Biol Chem, 2003, 278(6): 4135-4144.
[18] Sontheimer H. An unexpected role for ion channels in brain tumor metastasis [J]. Exp Biol Med (Maywood), 2008, 233(7): 779-791.
[19] Hockaday D C, Shen S, Fiveash J, et al. Imaging glioma extent with 131I-TM-601 [J]. J Nucl Med, 2005, 46(4): 580-586.
[20] Mamelak AN, Rosenfeld S, Bucholz R, et al. PhaseⅠsingle-dose study of intracavitary-administered iodine-131-TM-601 in adults with recurrent high-grade glioma [J]. J Clin Oncol, 2006, 24(22): 3644-3650.
[21] Haas BR, Sontheimer H. Inhibition of the sodium-potas-sium-chloride cotransporter isoform-1 reduces glioma invasion [J]. Cancer Res, 2010, 70(13): 5597-5606

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备注/Memo

备注/Memo:
作者单位:063000 河北唐山,华北理工大学附属唐山市人民医院(陆丽娟,李玉凤,赵喜庆,胡万宁)通讯作者:胡万宁,E-mail:rmyy_hwn@163.com
更新日期/Last Update: 1900-01-01